Is cramming the week's hard sessions into one block week better than spreading them?
Not here: 9 HIT in one week vs 3 a week for 3 weeks made no detectable difference in 20 junior skiers; recovery dipped in the block week.
McGawley and colleagues asked whether concentrating high-intensity interval training (HIT) into one week beats spreading it, inside the same polarized 3-week cycle.
Twenty well-trained junior cross-country skiers (10 women, 10 men) did both: EVEN, 3 HIT sessions a week for 3 weeks with easy training (LIT) around them, and BLOCK, all 9 HIT sessions in week 2 (twice a day on three days) with only LIT in weeks 1 and 3.
Every HIT session was 5 × 4 min of uphill diagonal-stride roller-skiing at the maximal sustainable intensity, with 6 min of active recovery; LIT time and strength sessions were matched (742 vs 754 min of LIT). The skiers covered the same average distance per interval in both (740 vs 736 m).
Changes in VO2max, skiing economy, 600-m time trial, salivary hormones, capillary density, fibre area and type, enzyme activities and VEGF and PGC-1α protein did not differ between EVEN and BLOCK; the 600-m time improved over the study regardless of intervention.
Perceived recovery before HIT sessions was lower in BLOCK (median 4 vs 6) and the recovery-stress state was lower after the loading week, and during BLOCK the female skiers' recovery-stress state was lower than the males' after week 2 and still after week 3 (15 vs 22, p = 0.014); the BLOCK-vs-EVEN differences were gone by the end (overall pREC and recovery-stress state not different).
Our reading: an independent, female-inclusive trial finding no evidence that blocking HIT is better, against a block literature that is otherwise mostly from one laboratory; it is small, short, end-of-season, and its BLOCK arm had no hard session in the week before testing — the point estimates for VO2max and economy leaned toward EVEN (effect sizes 0.67 and 0.79, not significant).
Protocol
Key Results
Pre-to-post change (Δ, mean ± SD) in each 3-week intervention (Table 3); the p value is for the difference in change, and the effect sizes carry no sign:
| Outcome | EVEN Δ | BLOCK Δ | p (EVEN vs BLOCK) | ES |
|---|---|---|---|---|
| VO2max (mL/kg/min) | +0.9 ± 2.6 | −0.8 ± 2.4 | 0.071 | 0.67 |
| Skiing economy (O2 L/min; lower is better) | −0.06 ± 0.10 (significant vs pre) | +0.02 ± 0.16 | 0.117 | 0.79 |
| 600-m time trial (s) | −3 ± 5 (significant vs pre) | −1 ± 6 | 0.280 | 0.44 |
| Resting IgA (μg/mL) | +40 ± 96 | −26 ± 69 | 0.081 | 0.68 |
| Capillary density (per mm², biopsy group) | +3 ± 34 | −20 ± 39 | 0.253 | 0.66 |
| HAD activity (μmol/min/g, biopsy group) | 0.0 ± 1.1 | −0.7 ± 1.0 (significant vs pre) | 0.317 | 0.60 |
| PGC-1α protein (AU, biopsy group) | +0.02 ± 0.02 (significant vs pre) | +0.03 ± 0.04 (significant vs pre) | 0.862 | 0.11 |
Mechanisms
Practical Application
Limitations
How this entry was checked and changed
controlled crossover with n = 20, read from the full text (open access, CC BY). The rubric places a crossover of 15 or more at B+; within B+ a weak one: the Methods do not describe the randomization and the order groups were unequal (13 vs 7, "for the logistical reason" of one training hill), there is no washout beyond the 4-day test period, each period lasted only 3 weeks, and the biopsy markers come from 11 skiers